I'm writing this from Borneo. We've just spent the morning at Sepilok orangutan sanctuary. We're a few days into a trip I've wanted to take with them for a long time.
I only see my parents a few times a year. They're approaching their 70s. This trip matters to me because I want lasting memories with them — and because I'm increasingly aware that those memories depend on something neither of them has thought about much: whether they can physically do the things that make memories possible.
This is a hard conversation. It's hard finally giving advice to the people who brought you up!
This piece is a version of the conversation I've been having with them over the last few days. It applies to most people's parents.
The gap between life and being able to live it
There's a useful distinction in the ageing literature between lifespan and healthspan. Lifespan is the years you're alive. Healthspan is the years you're functional — without significant disability, able to do the things that make life worth living.
The two numbers are not the same. According to the AIHW, an Australian female at age 65 can expect to live about another 22 years on average — but only around 9.5 of those years without any disability.1 Over half her remaining life will involve some level of disability. The figures for men are similar. That gap, on average, is the period spent managing chronic conditions, declining function, and increasing dependence.
This isn't an argument about extending life. The question is what the last decade of life looks like. Whether you spend it travelling, gardening, lifting your grandchildren, going to dinner with friends — or whether you spend it managing limitations.
The decisions that shape healthspan compound over decades. But the choices that shape your final ten to twenty years specifically — those are made now, in your 60s and 70s. Not by genetics. By what you do with your body, and what you don't.
What ageing looks like for most Australians
My parents are not outliers. They're typical. That's worth understanding before we get into specifics.
In 2022, 39% of Australian adults had hypertension. By age 55 to 64 that figure is 57%. By 75 and over it's 85%.2 My dad sits in the middle of that demographic. His high blood pressure and cholesterol are managed with medication. It sounds like nothing dramatic — but only because it is so common it has become the norm.
About 23% of Australian women aged 50 and over have osteoporosis when measured with bone density scans.3 The Geelong Osteoporosis Study found that by age 79 and older, 87% of women had osteoporosis at the spine, hip, or forearm.4 My mum sits within that range. Most of her friends would too if they were scanned.
Cardiovascular disease, hypertension, dyslipidaemia, osteoporosis, sarcopenia (age related muscle loss), and balance decline are the most common conditions of ageing in this country. They are the norm. Most of them respond meaningfully to exercise. The response curve is steep enough that the difference between an older adult who exercises and one who doesn't is enormous — measurable in physical function, fracture risk, falls risk, cognition, and mortality.
My mum and osteoporosis
My mum is the more active of my parents. She walks, she does some Pilates, she's not sedentary. From her perspective, she's doing exercise. From an evidence based perspective, she's not getting close to the dosage that meaningfully changes bone density.
The reason it matters is what happens when something goes wrong. If she trips on a Borneo boardwalk, the difference between catching herself and fracturing an ankle is a matter of strength, reaction time, and bone tolerance. And recovery from a fracture is not just about the bone knitting back together. It is about getting back on her feet, walking the dog, doing the stairs, returning to normal life. The strongest predictor of how well that recovery goes is what the body was doing before the fracture — the muscle, the fitness, the balance that had been built. People who arrive at a fracture deconditioned often never quite return to baseline.
Until recently, the standard advice for women with osteoporosis was caution. Avoid loading the spine. Don't lift heavy. Don't jump. The fear was that high load activity would cause fractures.
That advice has been overturned. The LIFTMOR trial, published by Watson and colleagues in the Journal of Bone and Mineral Research in 2018, randomised postmenopausal women with osteopenia and osteoporosis to either eight months of supervised high intensity resistance and impact training (five sets of five repetitions at over 85% of one repetition maximum — heavy by any standard) or a low intensity home program.5 The high intensity group improved bone mineral density at the spine and femoral neck. They improved functional performance. There were no adverse events.
The implication is uncomfortable but clear. The women most worried about loading their bones are often the women whose bones need loading the most. Bone responds to mechanical strain. Low intensity walking and Pilates do not generate strain anywhere near the threshold required to change bone density. Heavy resistance training does.
What my mum needs — and what most women in her position need — is not more cardio. It is two strength sessions per week, with selected impact loading appropriate to her bone status, at a load that genuinely stresses the bone. And here is the catch: what most women her age think is heavy is not heavy. The LIFTMOR protocol used loads at over 85% of one repetition maximum — the weight you can lift only five times before your form breaks. Not the 2kg dumbbells that get handed out in most over-60s classes. The difference between those two doses is the difference between maintaining bone and changing it.


